Alias: MSTN, Myostatin HMP
Purity: 98%min
Specification: 1mg
Storage: 2-8 degree centigrade refrigerator


What is the GDF 8 / Myostatin ?
Myostatin (also known as growth differentiation factor 8, abbreviated GDF-8) is a myokine, a protein produced and released by myocytes that acts on muscle cells’ autocrine function to inhibit myogenesis: muscle cell growth and differentiation. In humans it is encoded by the MSTN gene. Myostatin is a secreted growth differentiation factor that is a member of the TGF beta protein family.



1. Treat muscle atrophy, strengthen muscle toughness,Increase muscle number.
2. Treating malnutrition patients is an important index of the strength training athletes.
3. GDF-8 can effectively prevent the increased due to age brings the increase of adipose tissue, and reduce the occurrence of diabetes.


How does GDF-8 work?

Some studies show that the lack of myostatin gives rise to impressive results in the increase of muscle mass both in humans and in animals. In bones specifically, the lack of myostatin increases its mineral density, resulting in an increase of the calcium levels of the organism. GDF-8 is stored in bones and it is necessary for the correct functionality of the heart, muscles, and nerves as well as for blood clotting.
While using this recombinant myostatin propeptide, it was found that collagen-rich fibrous tissue was plentiful in the injured area of the individual’s treated muscle, which has as a result the improvement of the regenerative capacity of the body.
Myostatin deficiency increases the ostegenic potential (formation of bone tissue) of bone marrow. There have been some studies related to the healing properties of stem cells originated in the bone marrow that has an auto regeneration capacity. This benefit can be promoted by blocking Myostatin on humans.
Myostatin inhibitors are used on muscular dystrophy treatment in the attempt to accomplish the muscle regeneration. Myostatin propeptide enhances muscle regeneration, increases myofiber hypertrophy (an increase in size of skeletal muscle through a growth in size of its component cells) and decreases fibrosis (formation of excess fibrous connective tissue, which can obliterate the function of the affected limb) in muscle healing.